Supports the whole raison d'etre of Trick and Treat
On Monday 26 January 2009, UK's Daily Express published two stories that illustrate the sheer and utter incompetence and wastefulness of the present government and 'health' boffins. They also demonstrate how the 'health' industry is able to con the media so that they can profit from our ill-health.
Article 1:
NOW FOOD POLICE PLAN TO SWOOP ON YOUR FRIDGE
SQUADRONS of “Food Police” are to start knocking on doors to lecture families on how to feed themselves properly.
In a move branded “Government nannying at its worst”, the teams – operated by councils across the country – will be recruited to visit homes at meal times before handing out advice on diet and how to reduce waste.
Eight thousand Food Police, or Love Food Champions under their official title, will be paid up to £8.50 an hour of taxpayers’ cash. And if a pilot scheme is successful, the idea could be rolled out across the country, costing the taxpayer tens of millions of pounds.
Employed by a private contractor, the teams will advise householders on how to plan their shopping carefully so that they do not over-cater.
You can read the rest at: http://www.express.co.uk/posts/view/81804/Now-Food-Police-plan-to-swoop-on-your-fridge
Apart from the obvious intrusion into people's lives and the total waste of taxpayers' money (I thought we were in a recession and short of the stuff), what possible good will it do? Don't people know now that they are wasting money by buying more food than they can eat and so on?
And if these cretins think that advising people to eat more 'healthy' carb-based foods to cut the incidence of conditions such as obesity and diabetes, when there is such a huge amount of evidence that this adv ice is a major cause of these illnesses, then the result can only be that the situation will get worse.
Article 2:
OBESITY BUG YOU CAN CATCH
OBESITY can be “caught” as easily as a common cold from other people’s coughs, sneezes and dirty hands, scientists will claim today.
Researchers believe that an airborne “adenovirus” germ could be causing the fat plague that is blighting Britain and other countries.
As many as one in three obese people may have become overweight after falling victim to the highly infectious cold-like virus, known as AD-36.
Read the rest here: http://www.dailyexpress.co.uk/posts/view/81810
Even if this is true, which I doubt, increasing the numbers of fat cells (adipocytes) won't make you any fatter — unless you fill those adipocytes with fat!
Stories like these are making me lose the will to live (at least in this country!)
But there are two other points:
1: Just what qualifications will these £8.50 per hour 'advisors' have to tell me what to do?
2. As my doctor is cannot legally prescribe for me without my consent, what law allows the (probably unqualified) otherwise unemployed to tell me what I can and can't eat?
Studies and other evidence published since Trick and Treat went to press
26 January 2009
13 January 2009
Low-carb, high-protein diet beats low-fat diet for weight loss - again
Supports Chapter 4: Learning from history,
Chapter 19: 'Healthy eating' is fattening,
Chapter 20: Diabetes deceit, and
Chapter 21: Diseases of the heart and blood vessels
Results from a systematic review demonstrate, yet again, that low-carbohydrate, high-protein diets are more effective for reducing weight and improving cardiovascular health than are low-fat diets.
Catherine Rolland and colleagues from Robert Gordon University in Aberdeen, UK, carried out a systematic review of 13 randomized, controlled trials including a total of 1222 individuals comparing low-carbohydrate, high-protein diets with low-fat diets.
Inclusion criteria included publication between January 2000 and March 2007, length of at least 6 months, participants aged 18 years and above, and a mean or median body mass index of at least 28 kg/m2.
Of the 13 studies, five lasted for 6 months, six for 12 months, one for 17 months, and one for 36 months. Eleven studies compared low-carbohydrate, high-protein diets with low-fat, high-carbohydrate diets and two compared medium-protein with high-protein diets. Measurements were compared at 6 and 12 months.
At 6 months, weight loss was an average of 4.02 kg greater in the low-carbohydrate, high-protein group than in the low-fat diet group.
At 12 months, the difference between the two groups had reduced, with the low-carbohydrate, high-protein group having lost an average of 1.05 kg more than the low-fat group.
Rolland and team also noted improvements in serum lipids, although these were more mixed, as a significant improvement in high-density lipoprotein and triglycerides was seen favoring the low-carbohydrate, high-protein group up to 12 months, but improvements in total and low-density lipoprotein cholesterol were higher in the low-fat group at 6 months.
A nonsignificant trend toward improvement in diastolic and systolic blood pressure was also observed up to 17 months for the low-carbohydrate, high-protein group.
The researchers conclude in the journal Obesity Reviews that their results show low-carbohydrate, high-protein diets are more effective for weight loss up to 12 months than low-fat diets with unrestricted or high levels of carbohydrates.
They add that although trends toward cardiovascular improvement favoring the low-carbohydrate, high-protein diet were seen in this study, “more evidence and longer-term studies are needed to assess the long-term cardiovascular benefits from the weight loss achieved using these diets.”
But there have already been many studies, both clinical and epidemiologic dating back over 140 years.
For example, as I showed in Trick and Treat, a study conducted in 1932 with four different diets with the same number of calories but different constituents gave these results in grams of weight lost per day on average:
high-carbohydrate/low-fat diet – 49 grams (Typical 'slimming' diet)
high-carbohydrate/low-protein – 122 grams
low-carbohydrate/high-protein – 183 grams
low-carbohydrate/high-fat – 205 grams (As recommended in T&T)
Those were all 1,000 kcals. But obese patients also lost weight at 2,700-kcals - but only on the low-carb high fat diet.
How much more evidence will it take before the disastrous 'healthy eating' experiment is called to a close - and those perpetrating it, and who are responsible for the increasing ill-health in our society, are called to account?
Hession M, et al. Systematic review of randomized controlled trials of low-carbohydrate vs. low-fat/low-calorie diets in the management of obesity and its comorbidities. Obes Rev 2009; 10: 36-50
DOI: 10.1111/j.1467-789X.2008.00518.x
Chapter 19: 'Healthy eating' is fattening,
Chapter 20: Diabetes deceit, and
Chapter 21: Diseases of the heart and blood vessels
Results from a systematic review demonstrate, yet again, that low-carbohydrate, high-protein diets are more effective for reducing weight and improving cardiovascular health than are low-fat diets.
Catherine Rolland and colleagues from Robert Gordon University in Aberdeen, UK, carried out a systematic review of 13 randomized, controlled trials including a total of 1222 individuals comparing low-carbohydrate, high-protein diets with low-fat diets.
Inclusion criteria included publication between January 2000 and March 2007, length of at least 6 months, participants aged 18 years and above, and a mean or median body mass index of at least 28 kg/m2.
Of the 13 studies, five lasted for 6 months, six for 12 months, one for 17 months, and one for 36 months. Eleven studies compared low-carbohydrate, high-protein diets with low-fat, high-carbohydrate diets and two compared medium-protein with high-protein diets. Measurements were compared at 6 and 12 months.
At 6 months, weight loss was an average of 4.02 kg greater in the low-carbohydrate, high-protein group than in the low-fat diet group.
At 12 months, the difference between the two groups had reduced, with the low-carbohydrate, high-protein group having lost an average of 1.05 kg more than the low-fat group.
Rolland and team also noted improvements in serum lipids, although these were more mixed, as a significant improvement in high-density lipoprotein and triglycerides was seen favoring the low-carbohydrate, high-protein group up to 12 months, but improvements in total and low-density lipoprotein cholesterol were higher in the low-fat group at 6 months.
A nonsignificant trend toward improvement in diastolic and systolic blood pressure was also observed up to 17 months for the low-carbohydrate, high-protein group.
The researchers conclude in the journal Obesity Reviews that their results show low-carbohydrate, high-protein diets are more effective for weight loss up to 12 months than low-fat diets with unrestricted or high levels of carbohydrates.
They add that although trends toward cardiovascular improvement favoring the low-carbohydrate, high-protein diet were seen in this study, “more evidence and longer-term studies are needed to assess the long-term cardiovascular benefits from the weight loss achieved using these diets.”
But there have already been many studies, both clinical and epidemiologic dating back over 140 years.
For example, as I showed in Trick and Treat, a study conducted in 1932 with four different diets with the same number of calories but different constituents gave these results in grams of weight lost per day on average:
high-carbohydrate/low-fat diet – 49 grams (Typical 'slimming' diet)
high-carbohydrate/low-protein – 122 grams
low-carbohydrate/high-protein – 183 grams
low-carbohydrate/high-fat – 205 grams (As recommended in T&T)
Those were all 1,000 kcals. But obese patients also lost weight at 2,700-kcals - but only on the low-carb high fat diet.
How much more evidence will it take before the disastrous 'healthy eating' experiment is called to a close - and those perpetrating it, and who are responsible for the increasing ill-health in our society, are called to account?
Hession M, et al. Systematic review of randomized controlled trials of low-carbohydrate vs. low-fat/low-calorie diets in the management of obesity and its comorbidities. Obes Rev 2009; 10: 36-50
DOI: 10.1111/j.1467-789X.2008.00518.x
27 December 2008
Autism And Schizophrenia Share Common Origin
Supports Chapter 26: Diet and the brain
and Chapter 19: Prevention is better
Medical News Today has just reported a study conducted by a Dutch researcher, Annemie Ploeger, in which she hypothesises that "Schizophrenia and autism probably share a common origin".
There is already research which links these conditions to our 'healthy' diet in infancy, but what is new is that Ploeger also indicts "disruptions" to the fetus during the early growth period - between 20 and 40 days after fertilisation - when the embryo is highly susceptible to such disruptions. Such 'disruptions' she puts down to the mother taking a morning sickess drug called softenon. However, morning sickness is less likely if the pregnant mother is eating a natural diet.
If Ploeger is right, and I have every reason to suspect that she is, an expectant mother's diet as well as the diet her baby eats during its postnatal formative period, may both play an important role in the growing incidences of both autism and schizophrenia.
http://www.medicalnewstoday.com/articles/133293.php
and Chapter 19: Prevention is better
Medical News Today has just reported a study conducted by a Dutch researcher, Annemie Ploeger, in which she hypothesises that "Schizophrenia and autism probably share a common origin".
There is already research which links these conditions to our 'healthy' diet in infancy, but what is new is that Ploeger also indicts "disruptions" to the fetus during the early growth period - between 20 and 40 days after fertilisation - when the embryo is highly susceptible to such disruptions. Such 'disruptions' she puts down to the mother taking a morning sickess drug called softenon. However, morning sickness is less likely if the pregnant mother is eating a natural diet.
If Ploeger is right, and I have every reason to suspect that she is, an expectant mother's diet as well as the diet her baby eats during its postnatal formative period, may both play an important role in the growing incidences of both autism and schizophrenia.
http://www.medicalnewstoday.com/articles/133293.php
17 December 2008
Low cholesterol increases risk of bone fracture
Supports Chapter 22: The dangers of low cholesterol
A new study shows that low levels of cholesterol, LDL and triglycerides are associated with fractures of the vertebrae in postmenopausal women.
“Many factors other than low bone mineral density (BMD) have been suggested as predictors of risk for osteoporosis-related fractures,” comment Ebru Alemdaroglu and colleagues from Ankara Numune Training and Research Hospital, Turkey.
They add that growing evidence suggests bone and fat metabolism are related, but data are limited and contradictory.
To investigate the effect of the serum lipid levels on BMD and vertebral fractures, Alemdaroglu and team examined lumbar spine, hip and radius bone mineral density (BMD) measurements, lateral dorsal and lumbar spine radiographs, and serum lipid levels in 107 postmenopausal women aged 45–79 years.
The researchers were able to score 89 radiographs with good technical properties using the Kleerekoper method. Vertebrae fractures were observed in 71% of the women.
Analysis showed that patients with vertebrae fractures had significantly lower levels of total cholesterol, triglycerides and LDL cholesterol than the patients without vertebrae fractures.
Total cholesterol level was most strongly associated with vertebral fracture. An increase of 1 mg/dl (0.03 mmol/l) total cholesterol was associated with a 2.2% decreased risk for vertebrae fracture.
The researchers reason that estrogen is synthesized by cholesterol and esterified forms of estrogen are stored and transported by lipoproteins. Thus, decreased LDL levels would be associated with decreased stored estrogen and may explain the relationship between vertebrate fracture and reduced serum lipids.
According to the T-scores obtained by BMD measurement, 36 (33.6%) of the 107 women examined were suffering from osteoporosis. Alemdaroglu and co-researchers report that the lipid profiles of women with osteoporosis did not differ significantly from those without osteoporosis.
There was no correlation between serum lipid levels and BMD at the lumbar spine, right hip and radius in any of the study participants. Only total cholesterol and LDL cholesterol were weakly associated with BMD at the forearm after the adjustment for possible confounders.
Sivas F, et al. Serum lipid profile: its relationship with osteoporotic vertebrae fractures and bone mineral density in Turkish postmenopausal women. Rheumatol Int 2008. [Online publication ahead of print]
DOI 10.1007/s00296-008-0784-4
COMMENT
The bottom line is: This is yet another indication that low cholesterol levels are not desirable.
A new study shows that low levels of cholesterol, LDL and triglycerides are associated with fractures of the vertebrae in postmenopausal women.
“Many factors other than low bone mineral density (BMD) have been suggested as predictors of risk for osteoporosis-related fractures,” comment Ebru Alemdaroglu and colleagues from Ankara Numune Training and Research Hospital, Turkey.
They add that growing evidence suggests bone and fat metabolism are related, but data are limited and contradictory.
To investigate the effect of the serum lipid levels on BMD and vertebral fractures, Alemdaroglu and team examined lumbar spine, hip and radius bone mineral density (BMD) measurements, lateral dorsal and lumbar spine radiographs, and serum lipid levels in 107 postmenopausal women aged 45–79 years.
The researchers were able to score 89 radiographs with good technical properties using the Kleerekoper method. Vertebrae fractures were observed in 71% of the women.
Analysis showed that patients with vertebrae fractures had significantly lower levels of total cholesterol, triglycerides and LDL cholesterol than the patients without vertebrae fractures.
Total cholesterol level was most strongly associated with vertebral fracture. An increase of 1 mg/dl (0.03 mmol/l) total cholesterol was associated with a 2.2% decreased risk for vertebrae fracture.
The researchers reason that estrogen is synthesized by cholesterol and esterified forms of estrogen are stored and transported by lipoproteins. Thus, decreased LDL levels would be associated with decreased stored estrogen and may explain the relationship between vertebrate fracture and reduced serum lipids.
According to the T-scores obtained by BMD measurement, 36 (33.6%) of the 107 women examined were suffering from osteoporosis. Alemdaroglu and co-researchers report that the lipid profiles of women with osteoporosis did not differ significantly from those without osteoporosis.
There was no correlation between serum lipid levels and BMD at the lumbar spine, right hip and radius in any of the study participants. Only total cholesterol and LDL cholesterol were weakly associated with BMD at the forearm after the adjustment for possible confounders.
Sivas F, et al. Serum lipid profile: its relationship with osteoporotic vertebrae fractures and bone mineral density in Turkish postmenopausal women. Rheumatol Int 2008. [Online publication ahead of print]
DOI 10.1007/s00296-008-0784-4
COMMENT
The bottom line is: This is yet another indication that low cholesterol levels are not desirable.
12 December 2008
Yet more vitamin D deficiency diseases
Supports Chapter 11: Our irrational fear of sunlight
I really should have delayed Trick and Treat; there is so much more evidence coming out in support of its various chapters and subjects since it went to print in September.
Two studies just published show even more dangers of ill-health caused by the current ‘keep out of the sun’ advice. These concern low levels of vitamin and Parkinson’s disease, and the inflammatory bowel diseases, Crohn’s disease and ulcerative colitis.
Parkinson’s disease
A team of doctors at the Department of Neurology, Emory University School of Medicine, Atlanta, GA, compared the prevalence of vitamin D deficiency in mainly white patients with Parkinson’s disease, with the prevalence in age-matched healthy controls and patients with Alzheimer disease, between 1992 and 2007.
They found significantly lower levels of vitamin D levels at a mean of 31.9 nmol/l in the Parkinson’s patients compared to the other two groups.
(Alzheimer’s patients levels were also lower than the levels in the healthy cohort, although the study was not set up to measure the effects of this.)
Evatt ML, et al Prevalence of vitamin d insufficiency in patients with Parkinson disease and Alzheimer disease. Arch Neurol 2008; 65: 1348-52.
Inflammatory bowel disease
A causal connection between vitamin D deficiency and inflammatory bowel disease was reported at the Annual Scientific Meeting of the American College of Gastroenterology in Orlando, Florida, during October.
Lead researcher Dr Alex Ulitsky from the Medical College of Wisconsin in Milwaukee, USA, and his team found that Vitamin D deficiency was common among people with inflammatory bowel disease and is associated with increased disease activity and worse quality of life.
They found that nearly 50% of the patients were Vitamin D deficient at some point, with 11% being severely deficient. Vitamin D deficiency was also associated with reduced quality of life in patients with Crohn’s disease, but not in those with ulcerative colitis.
Although concerned mainly with Crohn’s disease and ulcerative colitis, Dr Ulitsky concluded: “All inflammatory bowel disease patients, irrespective of their disease, disease location or nature should have their Vitamin D levels checked regularly and corrected aggressively when insufficiency is found.”
Meeting website: http://www.acg.gi.org/acgmeetings/
COMMENT
There may be some excuse for people living at higher latitudes to have some vitamin D deficiency, particularly if they have darker skins, but for residents of Florida, with its almost year-long sunshine also to suffer must be a reflection on the appalling health advice they are given. We need to get out in the sun more, not less.
I really should have delayed Trick and Treat; there is so much more evidence coming out in support of its various chapters and subjects since it went to print in September.
Two studies just published show even more dangers of ill-health caused by the current ‘keep out of the sun’ advice. These concern low levels of vitamin and Parkinson’s disease, and the inflammatory bowel diseases, Crohn’s disease and ulcerative colitis.
Parkinson’s disease
A team of doctors at the Department of Neurology, Emory University School of Medicine, Atlanta, GA, compared the prevalence of vitamin D deficiency in mainly white patients with Parkinson’s disease, with the prevalence in age-matched healthy controls and patients with Alzheimer disease, between 1992 and 2007.
They found significantly lower levels of vitamin D levels at a mean of 31.9 nmol/l in the Parkinson’s patients compared to the other two groups.
(Alzheimer’s patients levels were also lower than the levels in the healthy cohort, although the study was not set up to measure the effects of this.)
Evatt ML, et al Prevalence of vitamin d insufficiency in patients with Parkinson disease and Alzheimer disease. Arch Neurol 2008; 65: 1348-52.
Inflammatory bowel disease
A causal connection between vitamin D deficiency and inflammatory bowel disease was reported at the Annual Scientific Meeting of the American College of Gastroenterology in Orlando, Florida, during October.
Lead researcher Dr Alex Ulitsky from the Medical College of Wisconsin in Milwaukee, USA, and his team found that Vitamin D deficiency was common among people with inflammatory bowel disease and is associated with increased disease activity and worse quality of life.
They found that nearly 50% of the patients were Vitamin D deficient at some point, with 11% being severely deficient. Vitamin D deficiency was also associated with reduced quality of life in patients with Crohn’s disease, but not in those with ulcerative colitis.
Although concerned mainly with Crohn’s disease and ulcerative colitis, Dr Ulitsky concluded: “All inflammatory bowel disease patients, irrespective of their disease, disease location or nature should have their Vitamin D levels checked regularly and corrected aggressively when insufficiency is found.”
Meeting website: http://www.acg.gi.org/acgmeetings/
COMMENT
There may be some excuse for people living at higher latitudes to have some vitamin D deficiency, particularly if they have darker skins, but for residents of Florida, with its almost year-long sunshine also to suffer must be a reflection on the appalling health advice they are given. We need to get out in the sun more, not less.
10 December 2008
Leading nutritionist shows how little she knows
Supports pretty well all of Trick and Treat
The UK's Daily Express published an article on Tuesday 9 December 2008 entitled "Did scientists get it wrong on the dangers of saturated fat?" It was written to publicise Trick and Treat: How 'healthy eating' is making us ill, but was in the form of a debate.
On the "Yes" side was me and my book, Trick and Treat. On the "No"side was an argument by a senior member of the British Nutrition Foundation, Dr Joanne Lunn. Her comments illustrate well why I felt it necessary to write Trick and Treat as she said that:
"The government, doctors and nutritionists don't base recommendations for reducing the amount of saturated fat in our diets on old research but on a growing body of evidence linking a diet high in saturated fat with a higher level of blood cholesterol and high blood cholesterol levels with a risk of cardiovascular disease."
The evidence I quoted in support of Trick and Treat is not 'old' evidence, but is right up-to-date; it includes studies published as recently as September this year. And that evidence shows over and over again that saturated fat does not cause cardiovascular diseases
Indeed, there has been so much evidence against 'healthy eating' since its inception in the 1980s that Professor Sylvan Lee Weinberg, a past President of the American College of Cardiology and a fervent supporter and advocate of 'healthy eating', finally wrote in the 4 March 2004 edition of the Journal of the American College of Cardiology, that:
Dr Lunn also said that "people will always ignore the evidence". But it is not I who am ignoring the evidence, it is people like Dr Lunn, and until those in authority stop ignoring the growing evidence that 'healthy eating' isn't healthy, our health can only deteriorate still further.
But, of course, if we didn't get ill, they wouldn't have a job, would they?
The UK's Daily Express published an article on Tuesday 9 December 2008 entitled "Did scientists get it wrong on the dangers of saturated fat?" It was written to publicise Trick and Treat: How 'healthy eating' is making us ill, but was in the form of a debate.
On the "Yes" side was me and my book, Trick and Treat. On the "No"side was an argument by a senior member of the British Nutrition Foundation, Dr Joanne Lunn. Her comments illustrate well why I felt it necessary to write Trick and Treat as she said that:
"The government, doctors and nutritionists don't base recommendations for reducing the amount of saturated fat in our diets on old research but on a growing body of evidence linking a diet high in saturated fat with a higher level of blood cholesterol and high blood cholesterol levels with a risk of cardiovascular disease."
The evidence I quoted in support of Trick and Treat is not 'old' evidence, but is right up-to-date; it includes studies published as recently as September this year. And that evidence shows over and over again that saturated fat does not cause cardiovascular diseases
Indeed, there has been so much evidence against 'healthy eating' since its inception in the 1980s that Professor Sylvan Lee Weinberg, a past President of the American College of Cardiology and a fervent supporter and advocate of 'healthy eating', finally wrote in the 4 March 2004 edition of the Journal of the American College of Cardiology, that:
"The low-fat, high-carbohydrate diet, promulgated vigorously . . . may well have played an unintended role in the current epidemics of obesity, lipid [blood fat] abnormalities, type II diabetes, and metabolic syndromes. This diet can no longer be defended by appeal to the authority of prestigious medical organizations or by rejecting clinical experience and a growing medical literature suggesting that the much-maligned low-carbohydrate, high-protein diet may have a salutary effect on the epidemics in question."Professor Weinberg is not alone; there is a growing number of doctors speaking out about the falsity of the current 'healthy' recommendations.
Dr Lunn also said that "people will always ignore the evidence". But it is not I who am ignoring the evidence, it is people like Dr Lunn, and until those in authority stop ignoring the growing evidence that 'healthy eating' isn't healthy, our health can only deteriorate still further.
But, of course, if we didn't get ill, they wouldn't have a job, would they?
03 December 2008
Study finds vegetarians have smaller brains
Supports Chapter 13: Homo carnivorous
Scientists at the Department of Physiology, Anatomy and Genetics, University of Oxford, have discovered that going veggie could be bad for your brain – with those on a meat-free diet six times more likely to suffer brain shrinkage.
The study involved tests and brain scans on community-dwelling volunteers aged 61 to 87 years without cognitive impairment at enrolment, over a period of five years. When the volunteers were retested five years later the medics found those with the lowest levels of vitamin B12 were also the most likely to have brain shrinkage. It confirms earlier research showing a link between brain atrophy and low levels of B12.
Vegans are the most likely to be deficient because the best sources of the vitamin are meat, particularly liver, milk and fish.
This study confirms other findings, covered in Trick and Treat, which shows that overall human brain sizes have reduced by an average 11% since we adopted an agricultural diet based on cereal grains rather than the meat-based diet of our Palaeolithic ancestors.
Vogiatzoglou A, et al. Vitamin B12 status and rate of brain volume loss in community-dwelling elderly. Neurology 2008; 71(11): 826-32.
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